3-way Tryst Goes Haywire Deadly – Microwaves are Beaming-in Demons

She admitted on-camera, on-scene, and in the moment that she and her ex-husband were fooling around in a naked 3-way tryst on her bed with a younger man she had recently transported up from California. The younger man went berserk and stabbed the ex-husband to death. The driving factors are of course far deeper and more sinister.

https://toxi.com/esmog

https://www.youtube.com/watch?v=q5kITpYMwUE

Womens’ brain chemistry, per Mother Nature design, makes them addictive to sex. It’s probably true for males also, but studies show women are addictable to sex via natural endorphins, opioids, etc.

20260131 — Personality flip — brain chemistry imbalance — violent takeover by unseen forces — is tell-tale of ‘real’ or ‘apparent’ “demonic” takeover. This is an area of high contention presently. **Regardless of true cause or causes, the overall effect is nightmarish.**

Wireless Gene Remote Control a dimmer switch for biology

Blasted by Media and Microwaves — “It’s like a wild animal.” – What’s going wrong with behavior in schools? – ‘Fk You Btch’: Cops Handcuff 9-Year-Old Throwing Tantrum at Florida Elementary School

Below are solid, citable sources that connect female sexual arousal/orgasm to reward neurochemistry, including endogenous opioids (endorphins/opioid receptors), plus a few clinical “compulsive/overuse” (addiction-like) papers where opioid antagonists (naltrexone) are used.

Table of Contents

Human studies in women that directly implicate endogenous opioids

Opioid blockade (naltrexone) reduces sexual responding in both women and men (randomized, placebo-controlled crossover; n=64, including 32 women). (PubMed)
Women: orgasm produces a neuroendocrine signature, while peripheral beta-endorphin does not rise (masturbation-induced orgasm; blood beta-endorphin unchanged, prolactin rises). This is often cited to show that “blood endorphins” are a poor proxy for what the brain’s opioid system is doing. (PubMed)

Female orgasm / arousal neuroimaging (reward circuitry engagement)

fMRI: “Brain Activity Unique to Orgasm in Women” (orgasm-related activation across cortical/subcortical/brainstem regions; widely cited). (PMC)
PET (rCBF): clitorally induced orgasm in healthy women; includes reward-relevant regions (e.g., ventral midbrain/caudate), interpreted as implicating dopamine and orgasm-related disinhibition patterns. (PubMed)
PET: female orgasm activates the pituitary (authors interpret as reflecting endocrine release relevant to orgasm physiology). (PubMed)

Female-animal evidence that opioids mediate “sexual reward” learning

Conditioned place preference from “paced mating” in female rats is opioid-dependent (naloxone blocks the mating-induced CPP), a classic demonstration that endogenous opioids are required for sexual reward conditioning in females. (PubMed)
Review: learned sexual behavior and the endogenous opioid system (covers how opioids modulate sexual reward/conditioning; useful for citing the mechanism). (PMC)
Review: female sexual behavior circuitry includes β-endorphin/opioid-linked hypothalamic pathways (mechanistic framing for how opioids gate receptivity and motivation circuits in females). (Frontiers)

“Addiction-like / compulsive” framing where opioid antagonists are used

Naltrexone augmentation for compulsive sexual behavior (clinical literature; includes discussion of opioid antagonism as a lever for compulsive sexual behaviors). (SAGE Journals)
Systematic review/case literature: naltrexone in hypersexuality (includes case-level evidence and mechanistic rationale via opioid receptors). (ResearchGate)

Direct in-vivo evidence of orgasm-triggered opioid release (not female, but highly relevant mechanism)

PET/fMRI shows endogenous opioid release after orgasm in humans (men)—this is one of the cleanest direct demonstrations that orgasm can trigger measurable μ-opioid–system activation in vivo. ([PMC][11])